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development of IAA was regarded as most likely due to oxidase deamination of NMT (Barker et al., 1980), but was later recognized being also in part by immediate deamination by MAO (Barker et al., 1982). Pretreatment Together with the MAO-I iproniazid in rat total brain homogenates inhibited IAA formation by eighty three%, NMT and DMT-NO formation h